Has it finally happened? Bird Flu death in China sparks fear of human-transmitted H5N1 strain
February 16, 2013 – CHINA – A woman diagnosed with the H5N1 strain of the bird flu last week, has died in southwest China. Health authorities in Guiyang, Guizhou province, announced that the 21-year-old woman, Shuai Pengyue, died on Wednesday due to multiple organ failure as a result of the flu. Shuai was one of two women reported in the area to have contracted the new strain of the avian influenza. Health officials have investigated the two of them and concluded that neither patient was in contact with poultry before showing symptoms of the illness. Victim proximity is important to note because typically, the bird flu is contracted by being in contact with poultry. In this case, health officials worry this could be signs that the H5N1 strain can now be transmitted between humans. Meanwhile, in Cambodia, a 3-year-old girl has become the sixth person to die from the bird flu in the country this year. The Cambodian Health Ministry and the World Health Organization released statements saying that the child was in contact with poultry recently in the southern province of Kampot. Cambodia has already reported seven human cases of the H5N1 virus this year, all of them fatal except one. Health officials and scientists have feared that the virus could mutate into a highly contagious strain which could be transmitted from human to human. Scientists in the Netherlands and the U.S. have been working on an artificially mutated version of the flu that is easily transmissible among humans in an attempt to do research for prevention or a cure. Research was halted until recently due to fears of a deadly global pandemic if the virus was accidentally removed from the controlled environment. Now, researchers are making a push to resume investigation of the deadly virus, especially in light of the new cases. Leo Poon Lit-man, an associate professor at the University Of Hong Kong School Of Public Health, told the South China Morning Post that he supports the controversial research. “The only way… to control the virus and come to a prevention plan is to allow the research to go forward,” Poon said. Adding, “the H5N1 is still a threat to humans, and it is true that the research may pose some risk. But we may also benefit from it, as we need further understanding of the virus to ensure a better response in case of an outbreak.” The mortality rate for the avian flu was as high as 60 percent during the 2003 outbreak in Southeast Asia. Most of the victims caught the disease from birds. – IBT
H5N1, the next pandemic? Scientists greatest fear was human transmission of the virus – 2008
SARS-like virus infects 3rd victim in UK: A third member of a family in the UK has been infected with a new respiratory illness similar to the deadly SARS virus, officials say. It strengthens evidence that the virus can spread between people, however experts say the risk to the general population remains small. Of the 12 people confirmed to have the virus, five have died. This case appears to be a milder form of the infection and the patient is not being treated in hospital. The infection is thought to come from contact with animals. However, if the virus can spread between people it poses a much more serious threat. This is the fourth case identified in the UK. The first was a patient flown in from Qatar for treatment. The second was linked to travel to the Middle East and Pakistan. The virus is then thought to have spread from the second patient to his son and another member of the family. Prof John Watson, head of the respiratory diseases department at the Health Protection Agency, said: “Although this patient had a mild form of respiratory illness, as a precaution the HPA is advising that the patient self-isolate and limit contact with non-household members. “Although this case appears to be due to person-to-person transmission, the risk of infection in contacts in most circumstances is still considered to be low.” Other people who came into contact with the family are being tested. –BBC
H5N1 found in Germany: About 14,000 ducks at a German farm are being slaughtered following a bird flu outbreak. A federal laboratory confirmed Friday the H5N1 virus was detected at the farm near Seelow, east of Berlin — the first such finding in Germany in more than three years. On Saturday, officials started slaughtering the farm’s ducks. Local council spokesman Tobias Seyfarth told news agency dpa that all poultry within a one-kilometer (half-mile) radius of the facility will be kept under observation for the next 21 days, with owners told to keep their birds where they are and report any symptoms. The H5N1 virus normally spreads between sick poultry, but it can sometimes spread from poultry to humans. Bird flu has killed 367 people worldwide since surfacing in 2003, the World Health Organization says. –SF Gate
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5 Amazing Properties of Sunlight You’ve Never Heard About
Sunlight is well-known to provide us vitamin D, but did you know that it kills pain, keeps us alert at night, burns fat and more…
Our biological connection and dependence to the sun is so profound, that the very variation in human skin color from African, melanin-saturated dark skin, to the relatively melanin de-pigmented, Caucasian lighter-skin, is a byproduct of the offspring of our last common ancestor from Africa (as determined by mitochondrial DNA) migrating towards sunlight-impoverished higher latitudes, which began approximately 60,000 years ago. In order to compensate for the lower availability of sunlight, the body rapidly adjusted, essentially requiring the removal of the natural “sunscreen” melanin from the skin, which interferes with vitamin D production; vitamin D, of course, is involved in the regulation of over 2,000 genes, and therefore is more like a hormone, without which our entire genetic infrastructure becomes destabilized.
While the health benefits of vitamin D are well-documented (GreenMedInfo.com has identified over 200 health conditions that may benefit from optimizing vitamin D levels: Vitamin D Health Benefits page, and Henry Lahore’s Vitamin D Wiki has far more), the therapeutic properties of sunlight are only now being explored in greater depth by the research community.
Below are detailed five noteworthy properties of sunlight exposure:
1) Sunlight Has Pain-Killing (Analgesic) Properties: A 2005 study published in the journal Psychosomatic Medicine titled, “The effect of sunlight on postoperative analgesic medication use: a prospective study of patients undergoing spinal surgery,” analyzed patients staying on the bright side of the hospital unit who were exposed to 46% higher-intensity sunlight on average. The patients exposed to an increased intensity of sunlight experienced less perceived stress, marginally less, took 22% less analgesic medication per hour, and had 21% less pain medication costs. [i]
2) Sunlight Burns Fat: A 2011 study published in The Journal of Investigative Dermatology revealed a remarkable fact of metabolism: The exposure of human skin to UV light results in increased subcutaneous fat metabolism. While subcutaneous fat, unlike visceral fat, is not considered a risk factor for cardiovascular disease, it is known that a deficiency of one of sunlight’s best known beneficial byproducts, vitamin D, is associated with greater visceral fat.[ii] Also, there is a solid body of research showing that vitamin D deficiency is linked to obesity, with 9 such studies on our obesity research page.
One of them, titled “Association of plasma vitamin D levels with adiposity in Hispanic and African Americans,” and which was published in the journal Anticancer Research in 2005, found that vitamin D levels were inversely associated with adiposity in Hispanics and African-Americans, including abdominal obesity.[iii] The point? Exposure to UVB radiation, which is most abundant two hours on either side of solar noon and responsible for producing vitamin D, may be an essential strategy in burning fat, the natural way.
3) Sunlight via Solar Cycles May Directly Regulate Human Lifespan: Published in 2010 in the journal Medical Hypotheses and titled, “The effect of solar cycles on human lifespan in the 50 United states: variation in light affects the human genome,” researchers review the possibility that solar cycles directly affect the human genome. According to the researchers:
In the current study we report that those persons conceived and likely born during the peaks (MAX approximately 3 years) of approximately 11-year solar cycles lived an average 1.7 years less than those conceived and likely born during non-peaks (MIN approximately 8 years). Increased energy at solar MAX, albeit relatively a small 0.1% increase from MIN, apparently modifies the human genome/epigenome and engenders changes that predispose to various diseases, thereby shortening lifespan. It is likely that same energy increases beneficial variety in the genome which may enhance adaptability in a changing environment.
Sunlight exposure, therefore, may directly affect the length of our life, and may even accelerate genetic changes that may confer a survival advantage.[iv]
4) Daytime Sunlight Exposure Improves Evening Alertness: A 2012 study published in the journal Behavioral Neuroscience titled, “Effects of prior light exposure on early evening performance, subjective sleepiness, and hormonal secretion,” found that subjects felt significantly more alert at the beginning of the evening after being exposed to 6 hours of mainly daylight exposure, whereas they became sleepier at the end of the evening after artificial light exposure.[v]
5) Sunlight May Convert To Metabolic Energy: If a novel hypothesis published in 2008 in the Journal of Alternative and Complementary Medicine is correct,[vi] a longstanding assumption that animals are incapable of utilizing light energy directly is now called into question. In other words, our skin may contain the equivalent of melanin “solar-panels,” and it may be possible to “ingest” energy, as plants do, directly from the Sun.
Melanin has a diverse set of roles in various organisms. From the ink of the octopus, to the melanin-based protective colorings of bacteria and fungi, melanin offers protection against a variety of threats: from predators and similar biochemical threats (host defenses against invading organisms), UV light, and other chemical stresses (i.e. heavy metals and oxidizing agents). Commonly overlooked, however, is melanin’s ability to convert gamma and ultraviolet radiation into metabolic energy within living systems.
Single-celled fungi, for instance, have been observed thriving within the collapsed nuclear reactor at Chernobyl, Ukraine, using gamma radiation as a source of energy. Albino fungi, without melanin, were studied to be incapable of using gamma radiation in this way, proving that gamma rays initiate a yet-unknown process of energy production within exposed melanin.
Vertebrate animals may also convert light directly into metabolic energy through the help of melanin. In a review titled, “Melanin directly converts light for vertebrate metabolic use: heuristic thoughts on birds, Icarus and dark human skin,” Geoffrey Goodman and Dani Bercovich offer a thought-provoking reflection on the topic, the abstract of which is well worth reading in its entirety:
Pigments serve many visually obvious animal functions (e.g. hair, skin, eyes, feathers, scales). One is ‘melanin’, unusual in an absorption across the UV-visual spectrum which is controversial. Any polymer or macro-structure of melanin monomers is ‘melanin’. Its roles derive from complex structural and physical-chemical properties e.g. semiconductor, stable radical, conductor, free radical scavenger, charge-transfer.
Clinicians and researchers are well acquainted with melanin in skin and ocular pathologies and now increasingly are with internal, melanized, pathology-associated sites not obviously subject to light radiation (e.g. brain, cochlea). At both types of sites some findings puzzle: positive and negative neuromelanin effects in Parkinsons; unexpected melanocyte action in the cochlea, in deafness; melanin reduces DNA damage, but can promote melanoma; in melanotic cells, mitochondrial number was 83% less, respiration down 30%, but development similar to normal amelanotic cells.
A little known, avian anatomical conundrum may help resolve melanin paradoxes. One of many unique adaptations to flight, the pecten, strange intra-ocular organ with unresolved function(s), is much enlarged and heavily melanized in birds fighting gravity, hypoxia, thirst and hunger during long-distance, frequently sub-zero, non-stop migration. The pecten may help cope with energy and nutrient needs under extreme conditions, by a marginal but critical, melanin-initiated conversion of light to metabolic energy, coupled to local metabolite recycling.
Similarly in Central Africa, reduction in body hair and melanin increase may also have lead to ‘photomelanometabolism’ which, though small scale/ unit body area, in total may have enabled a sharply increased development of the energy-hungry cortex and enhanced human survival generally. Animal inability to utilize light energy directly has been traditionally assumed. Melanin and the pecten may have unexpected lessons also for human physiology and medicine.
Source: http://www.greenmedinfo.com
- [i] Jeffrey M Walch, Bruce S Rabin, Richard Day, Jessica N Williams, Krissy Choi, James D Kang. The effect of sunlight on postoperative analgesic medication use: a prospective study of patients undergoing spinal surgery. Psychosom Med. 2005 Jan-Feb;67(1):156-63. PMID: 15673638
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New MRSA superbug strain found in UK milk supply
A new strain of MRSA has been found in British milk, indicating that the superbug is spreading through the livestock population and poses a growing threat to human health.
The new strain, MRSA ST398, has been identified in seven samples of bulk milk from five different farms in England.
The discovery, from tests on 1,500 samples, indicates that antibiotic-resistant organisms are gaining an increasing hold in the dairy industry.
The disclosure comes amid growing concern over the use of modern antibiotics on British farms, driven by price pressure imposed by the big supermarket chains. Intensive farming with thousands of animals raised in cramped conditions means infections spread faster and the need for antibiotics is consequently greater.
Three classes of antibiotics rated as “critically important to human medicine” by the World Health Organisation – cephalosporins, fluoroquinolones and macrolides – have increased in use in the animal population by eightfold in the last decade.
The more antibiotics are used, the greater the likelihood that antibiotic-resistant bacteria, such as MRSA, will evolve.
Experts say there is no risk of MRSA infection to consumers of milk or dairy products so long as the milk is pasteurised. The risk comes from farmworkers, vets and abattoir workers, who may become infected through contact with livestock and transmit the bug to others.
The discovery was made by scientists from Cambridge University who first identified MRSA in milk in 2011. They say the latest finding of a different strain is worrying.
Mark Holmes, of the department of veterinary medicine, who led the study, published in Eurosurveillance, said: “This is definitely a worsening situation. In 2011 when we first found MRSA in farm animals, the Department of Environment, Food and Rural Affairs [Defra] initially didn’t believe it. They said we don’t have MRSA in the dairy industry in this country.”
“Now we definitely have MRSA in livestock. What is curious is that it has turned up in dairy cows when in other countries on the Continent it is principally in pigs. Could it be in pigs or poultry in this country? We don’t know.”
The MRSA superbug can cause serious infections in humans which are difficult to treat, require stronger antibiotics, and take longer to resolve. Human cases of infection with the new strain have been found in Scotland and northern England according to Defra, but no details are available.
Dr Holmes said supermarket pressure on farmers to hold down prices was leading to the overuse of antibiotics to prevent cattle getting mastitis, an infection of the udder, that might interrupt the milk supply.
“If farmers were not screwed into the ground by the supermarkets and allowed to get a fair price for their milk they would be able to use fewer antibiotics,” he said.
“Common sense tells us that anything we can do to reduce use of antibiotics will reduce the growth of resistant bugs. We want to wean our farmers off antibiotics and the only way we can do that is with better regulation.”
Vets in Norway and Denmark had much more limited prescribing powers than in the UK, he added.
Source: http://www.independent.co.uk
For more information: http://lifelinknet.com
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New York Times Admits Stanford Organic Food Study Flaws, Apologizes
We broke the story of Stanford’s ridiculous organic food study the very night of its publication.
Now, a month later, the media is catching on to the study’s flaws; New York Times Opinion columnist Mark Bittman apologized for hoping—in vain—that the study would have little impact on the media.
“That was dumb of me,” he says, “and I’m sorry.”
Narrow Definitions and Egregious Oversights
The study suggests that organic animal and plant products are no healthier than conventionally grown varieties.
Bittman puts it beautifully:
“By providing ‘useful’ and ‘counterintuitive’ information about organic food, [the study authors] played right into the hands of the news hungry while conveniently obscuring important features of organic agriculture.”
The study authors narrowly—and misleadingly—defined the word “nutritious” and “healthy,” and on numerous occasions contradicted themselves.
How can food that the authors admit “may reduce exposure to pesticide residues and antibiotic-resistant bacteria” also be no more or less healthy than foods that don’t?
How can foods that put less waste and toxins into the groundwater be no more or less healthy for us than foods that pollute our water and harm those—animals and human—who drink it? We already know of the many nasty effects of pesticides and dangers of GMOs.
Even within its narrow constructs, the study authors erred. Newcastle University researcher Kirsten Brandt last year published a similar analysis of studies to conclude that organic foods do contain more nutrients.
How did Stanford miss this? By misspelling a critical class of nutrients found in produce that changed the results of the research: flavonols.
Accusations of Elitism Misplaced
Shortly after the study’s publication, fellow New York Times writer Roger Cohen “cheered” for its results and collectively called organic consumers narcissists.
He does admit that organic farming is “probably better for the environment,” but crucifies it for being “an elitist, pseudoscientific indulgence shot through with hype.”
Cohen’s accusation may be true for the likes of Whole Foods executives, who scream “buy organic” while filling their pretty stores with GMO-laden or GMO-supporting foods like Larabar, owned by General Mills, which has contributed over $500,000 to defeat Proposition 37. (If you haven’t seen the undercover ‘Organic Spies’ video about Whole Foods and GMOs, check it out in the past link.)
But the truth is this: organic farming is better for the earth and better for us.
The cost of this can be quite high; that’s why we must join together to promote organic, biodynamic farming across the globe rather than subjecting farmers in Africa to Monsanto corn, debt, and health problems. Monsanto doesn’t care about underprivileged farmers.
They care about lining their pockets.
Organic living should be available to all of us. It may seem like pie in the sky to some, but it isn’t. And we certainly shouldn’t be stopping the revolution.
To top it off, it’s worth noting that Stanford may have downplayed the benefits of food in response to a hefty donation from Cargill, the biggest private company in the US.
Although connections to a political or financial body may not indicate guilt, it’s certainly not helping.
So, who’s elitist now?
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Green Tea Compound Shows Promise for Tackling Cancer
A compound found in green tea could be a weapon in treatments for tackling cancer, according to newly published research at the University of Strathclyde.
The extract, known as epigallocatechin gallate (EGCG), has been known to have preventative anti-cancer properties but fails to reach tumours when delivered by conventional intravenous administration.
However, in initial laboratory tests at the Universities of Strathclyde and Glasgow, researchers used an approach which allowed the treatment to be delivered specifically to the tumours after intravenous administration. Nearly two-thirds of the tumours it was delivered to either shrank or disappeared within one month and the treatment displayed no side effects to normal tissues.
The tests are thought to be the first time that this type of treatment has made cancerous tumours shrink or vanish.
In the tests, on two different types of skin cancer, 40% of both types of tumour vanished, while 30% of one and 20% of another shrank. A further 10% of one of the types were stabilised.
The researchers encapsulated the green tea extract in vesicles that also carried transferrin, a plasma protein which transports iron through the blood. Receptors for transferrin are found in large amounts in many cancers.
Dr Christine Dufès, a senior lecturer at the Strathclyde Institute of Pharmacy and Biomedical Sciences, led the research. She said: “These are very encouraging results which we hope could pave the way for new and effective cancer treatments.
“When we used our method, the green tea extract reduced the size of many of the tumours every day, in some cases removing them altogether. By contrast, the extract had no effect at all when it was delivered by other means, as every one of these tumours continued to grow.
“This research could open doors to new treatments for what is still one of the biggest killer diseases in many countries.”
The research paper has been published in the journal Nanomedicine. Imaging equipment used in the research was funded by a grant from the Wellcome Trust.
The research links to Advanced Science and Technology, one of the research themes at Strathclyde’s Technology and Innovation Centre, a world-class facility uniting academic and industrial partners in seeking innovative research solutions, job creation and business development.
Source: Science Daily
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If You Wonder Why Medical CURES Are So Rare….
Type 1 Diabetes is a nasty disease in which the islet cells in the pancreas are destroyed by the body. This renders the sufferer unable to produce insulin, which is necessary to regulate the metabolism of glucose (sugar), the body’s main fuel.
Without insulin you will die. Type 1 diabetics must therefore inject insulin, either through the use of a pump or manually using syringes, literally through the day and manually test and regulate the blood sugar — a process that healthy people’s bodies do automatically.
It has been assumed that this condition, once it developed, was essentially incurable, although there were some people who believed that stem cells might be able to be “turned on” to replace the destroyed islet cells, or that we might some day come up with an artificial pancreas.
What I bet nobody assumed was that a relatively inexpensive, and 90 year old vaccine might actually cause the body to repair itself.
It looks like that may have been discovered.
A tuberculosis vaccine that has been in use for 90 years may help reverse Type 1 diabetes and eliminate the life-long need for insulin injections, results from an early study by Harvard University researchers suggest.
…
“These patients have been told their pancreases were dead,” said Denise Faustman, director of Harvard-affiliated Massachusetts General Hospital’s immunobiology laboratory, who led the study. “We can take those people, give them a very low dose twice and see their pancreases kick in and start to make small amounts of insulin.”
This is the sort of breakthrough — the possibility of an actual cure for a condition that has always been regarded as utterly incurable — that you would think everyone would be jumping all over to test and develop, right?
Wrong.
And why not?
Faustman and her colleagues at Massachusetts General inBoston are working to get the vaccine to market. After their early findings in studies with mice, she said they tried to interest every major drugmaker in developing the vaccine as a possible cure for diabetes. All told her there wasn’t enough money to be made in a cure that used an inexpensive, generically available vaccine, Faustman said.
Got it?
It’s not about people or health.
It’s about money.
The implication, of course, is that if it’s more profitable to “control” a disease than cure it, the path that will be pursued is “control”, not cure.
Are all these chronic conditions that we suffer from truly incurable?
Or is it simply that nobody looks for actual cures, because it doesn’t make as much money as “therapies” do?
It was a bit different when charities were providing a good part of the care. Then you had people actually interested in cures, because the funds were privately provided and the more people you could cure the more people you could help.
EMTALA and the rest of the mess our government put in place in the health system broke the incentives that would normally be associated with medicine, and we all get screwed as a consequence.
Source: http://market-ticker.org/
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So Just What Is Disease? A Grand Unified Theory
Does the common use of the word “disease” bypass critical reasoning to effectively mislead scientific thought? Modern medicine describes hundreds of individual “diseases.” Each “disease” is generally named for a group of symptoms and the area of the body affected. Many are suffixed by -itis, meaning inflammation of… like tonsill-itis or arthr-itis. And interestingly, recent studies are finding inflammation involved in virtually all of them. Does this terminology serve to pigeonhole distinct “diseases” and distract us from seeing the big picture of inflammation as the disease? Have we been looking at secondary pathologies and opportunistic microbes, and treating them at symptom-level… instead of addressing a common root cause? Is there some imaginary partition that separates human health from chemistry, physics and cell biology?
Whether inflammation is acute like appendicitis or chronic like atherosclerosis and obesity, an immune response is taking place. In-flam-mation literally means “on fire” and is classically marked by the Latin: rubor, tumor, calor and dolor — or redness, swelling, heat and pain — so we know from those words that oxidation is at work.
Oxidation is simply fire or rust or whenever one molecule seizes an electron from another molecule. The needy oxidant grabs or shares the electrons of an electron rich anti-oxidant. When the electrons are stolen from chemical bonds, those molecules (like DNA) come apart or are deformed (like fats) and said to be oxidized, burnt.
Inflammation does not just happen; a bacterium or toxin or some other irritant triggers an immune response. The ammunition used by the body for immune firefights is singlet oxygen, an all-purpose defensive weapon. With an unpaired electron, an oxygen radical is a powerful oxidant.
It can deconstruct and destroy pathogens, poisons, cell debris and other unwanted substances, molecule by molecule, by snatching the electrons that hold them together. Immune cells initiate the conflagration, armed with mini-flamethrowers that generate oxidative bursts of singlet oxygen to burn the area clean.
Mounting an oxidative immune response is dependent on the strength of the body’s electrochemical charge, which in turn hinges on a delicate balance between oxidants/acids (both electron-hungry) and overall body charge. (This charge is generated in mitochondria and at fluid/membrane interfaces. It is stored in fatty membranes, “anti-oxidant” molecules and water, and even flows from the Earth itself. Charge is transmitted via nerves and connective tissue meridians. If a potato can generate electricity, so can your body!) In the end, it is the electrochemical charge terrain that determines biochemical reactions, molecular integrity and health.
An oxidative immune response must be powered with, and yet contained by, abundant electrons, or damage will result to the cells it is designed to protect. To halt inflammation, the cause of the immune response needs to be removed, and then the flames of the defensive assault itself must be extinguished so charge can rise to normal and cells can be repaired or replaced.
So if we equate disease to inflammation, and understand that inflammation is oxidation, then we can understand that the common denominator of “disease” is oxidative stress — chronic unrelenting “fire” — loss of electrons, DNA dysfunction, and molecular and cellular deterioration.
Nearly undetectable low-level oxidative stress takes place even in healthy individuals and has become a focus of longevity research. Some of the most promising studies are concerned with the integrity of telomeres and mitochondrial membranes that take a beating, being at the center of metabolic oxidation and electron energy production. Fatty cell and mitochondrial membranes are prone to destruction when improper fats are consumed — making an All-American low-fat, high-glycemic, high-grain-carb/sugar/vegetable oil diet a train-wreck in progress.
Impaired membranes cannot transport oxygen or other substances to the cell, nor can they hold electron charge. These conditions bring disease, invite pathogens, cause cells to revert to cancerous anaerobic state, and eventually lead to cell death by suffocation. Undamaged Omega fats and varied saturated fats like butter and coconut oil are essential; crooked hydrogenated/oxidized/trans-fats/easily-oxidized polyunsaturated oils and a grain-based diet kill.
So are there individual “diseases” or just varied symptoms of inflammation expressing at the weakest seams in the boat — the points of nutritional inadequacy, sites of infection, trauma or toxic metal concentration or areas of circulatory stagnation, low charge and poor oxygenation? It all depends on how you look at it — on what level causes are sought.
We need oxygen to produce energy (electrons) and we need (electron) energy to deliver oxygen to mitochondria as a basic necessity of life. When net electron charge falls below optimum levels, oxygen/energy needs are unmet, inner balances shift toward acidity, hypoxia, oxidative stress, inflammation — and there is your disease.
What to do? Examine the three major areas of concern: nutrition, toxins/radiations and emotional states/lifestyle choices.
Nutrition can easily be divided into acid-forming and alkalizing food groups. A healthy balance favors alkalinity since acids rob electrons and charge. Processing strips electrons from foods. Fats are critical to membrane construction, should be consumed in pristine un-oxidized form and should be protected by fat-soluble antioxidants like Vitamins A, D, E and K and astaxanthin. Nutrition includes pH buffering electrolyte minerals like potassium and magnesium, and enzyme/hormone essentials like zinc, selenium and iodine, all of which are egregiously deficient across the population.
Nutrition also feeds gut bacteria and determines populations. These symbiants are there for our benefit and deserve respect.
Nutrients can be rated on the ORAC scale (oxygen radical absorbance capacity), so high ORAC means high electron content, antioxidant power. Look at cayenne and oregano, and small red beans and pecans, and direct your choices accordingly.
Nutrition is like the list of parts for a car: you need them all, and then to make it all work, the battery must have strong voltage. To make your biochemistry and DNA work, you need volts, too!
Toxins, metals and radiation all have one thing in common: they steal charge by generating oxidizing free radicals. They also trigger an immune response that brings inflammation. Detox! Organic toxins can be destroyed by oxidation. Pathogen attractive positively-charged metals can be reduced, mobilized and excreted by raising charge, supplementing proteolytic enzymes and supplying chelators like chlorella. Radiation damage can be prevented or ameliorated with antioxidant electrons that scrub free radicals formed of damaged cell molecules.
The mind is like the computer in the car. It monitors and adjusts internal conditions while the conscious part in the driver’s seat sets the tone of being. When the mood is chronically fearful, internal conditions deteriorate. When spirits are sunny and loving, your cells sing and your lifestyle choices become proactive — especially with healthy meals, exercise, breathing and physical activity at beneficial levels.
Check previous articles like “Healer’s Toolbox” for some of the substances that work if employed correctly and quantitatively, giving this approach a practical validity and logic. To reverse disease, the sources of inflammation must be oxidized/burned/removed and then the flames of inflammation snuffed out by an overwhelming electron boost to restore charge/voltage to operational levels and obtain sufficient oxygen to generate more charge and reverse damage. Hundreds of specific vitamins, minerals, phytonutrients, enzymes, amino acids and fats have proven useful in studies covering a wide range of maladies ultimately boosting body voltage and returning internal conditions to viable parameters.
Perhaps we should reevaluate the words at the foundation of health science. What if “diseases” are just symptoms? What if disease is simply inflammation, weak voltage—-oxidative stress? Maybe an artful and quantitative application of these forbidden principles should be at the foundation of healing.
Source: greenmedinfo.com
Thursday, August 2nd 2012 at 5:00 am by Capt. Randall
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